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This section of the review will describe how VEGFR-2 acts as a mechanoreceptor and closely interacts with other well-characterized mechanotransductive factors, demonstrating that altered physical forces caused by disease states could affect angiogenesis
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These findings support the role of iron-induced death in Mtb-triggered necrosis
MSCs cultured on GAG-mimetic peptide nanofibers formed cartilage-like nodules and deposited cartilage-specific matrix components
